43 HYPERURICEMIA, GOUT AND IDIOPATHIC ASEPTIC NECROSIS OF BONE
نویسندگان
چکیده
منابع مشابه
Introduction: hyperuricemia and gout.
The pathophysiology of gout is well understood, and effective treatments for acute gout and hyperuricemia leading to gout are widely available. Nonetheless, many patients suffer from severe tophaceous gouty arthritis, adverse effects of medications for gout, and inadequate treatment of hyperuricemia. The incidence of gout is rising in both men and women. The diagnosis and treatment of gout are ...
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This paper reports a detailed examination of three lesions of aseptic necrosis of bone occurring in a man who had worked in compressed air eleven years previously. Although two of the lesions were juxta-articular, in neither had the bone collapsed. Histology of only one similar lesion is reported in the literature (McCallum Ct a!. 1966). Necropsy material is considered, together with radiograph...
متن کاملAseptic necrosis of bone in caisson disease.
Caisson disease, or decompression sickness, is encountered when a raised atmospheric pressure on the body is reduced too rapidly and excess gases which have been held in solution in the blood and tissue fluids are set free as their solubility decreases. Typically the condition is manifested by tingling and paraesthesia, itching of the skin, and by bone and joint pains. The symptoms vary within ...
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In the past few decades, gout has increased not only in prevalence, but also in clinical complexity, the latter accentuated in part by a dearth of novel advances in treatments for hyperuricemia and gouty arthritis. Fortunately, recent research reviewed here, much of it founded on elegant translational studies of the past decade, highlights how gout can be better managed with cost-effective, wel...
متن کاملManagement of hyperuricemia and gout in CKD.
A 55-year-old man with history of kidney transplantation years ago because of diabetic nephropathy has severe ophaceous gout. He has been unable to tolerate treatment ith allopurinol because of worsening skin rashes. His last erum creatinine level was 2.3 mg/dL (203.3 mol/L). urrent medications include tacrolimus, mycophenolate ofetil, and prednisone at a dose of 20 mg/d orally. He resents to t...
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ژورنال
عنوان ژورنال: Pediatric Research
سال: 1988
ISSN: 0031-3998,1530-0447
DOI: 10.1203/00006450-198807000-00067